[2] MEIJERS B K, EVENEPOEL P. The gut-kidney axis: indoxyl sulfate, p-cresyl sulfate and CKD progression[J]. Nephrol Dial Transplant,2011,26(3):759-761.
[3] RITZ E. Intestinal-renal syndrome:mirage or Reality?[J]. Blood Purification, 2011, 31(1-3): 70-76.
[7] SABATINO A, REGOLISTI G, BRUSASCO I, et al. Alterations of intestinal barrier and microbiota in chronic kidney disease[J]. Nephrol Dial Transplant, 2015, 30(6): 924-933.
[8] FIACCADORI E, COSOLA C, SABATINO A. Targeting the gut for early diagnosis, prevention, and cure of diabetic kidney disease: is the phenyl sulfate story another step forward?[J]. Am J Kidney Dis, 2020, 75(1): 144-147.
[9] SUN G, YIN Z, LIU N, et al. Gut microbial metabolite TMAO contributes to renal dysfunction in a mouse model of diet-induced obesity[J]. Biochem Biophys Res Commun,2017,493(2):964-970.
[12] PIENIAZEK A, GWOZDZINSKI L, HIKISZ P,et al. Indoxyl sulfate generates free radicals, decreases antioxidant defense, and leads to damage to mononuclear blood cells[J]. Chem Res Toxicol,2018,31(9): 869-875.
[13] LIU W C, TOMINO Y, LU K C. Impacts of indoxyl sulfate and p-cresol sulfate on chronic kidney disease and mitigating effects of AST-120[J]. Toxins, 2018, 10(9): 367.
[18] CHENG X, ZHOU T, HE Y, et al. The role and mechanism of butyrate in the prevention and treatment of diabetic kidney disease[J]. Front Microbiol,2022,13:961536.